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An improved comprehension of the free radical biology mediating GI disease is essential for developing effective future treatments to reduce intestinal inflammation and improve the quality of life in affected individuals

Experientia 35 , 128130 (1979)

We also tested the responses of LCS3-sensitive and LCS3-resistant cell lines to C1-27 and did not observe any differential sensitivity to C1-27 among cell lines (Supplementary Fig

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