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The Gpx4 BI-KO mouse was generated by a conditional deletion of Gpx4 in forebrain neurons by administration of tamoxifen

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doi:10.1038/nature09165

According to flow cytometry analysis, this combination of treatments caused HCT-116 cells to undergo G0/G1 cell cycle arrest, elevate reactive oxygen species (ROS) levels, encourage apoptosis, and lose their mitochondrial membrane potential