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The former refers to the prevention of AF by controlling risk factors such as diabetes, hypertension, and heart failure before AF occurs, while the latter refers to reducing or reversing atrial electrical and structural remodeling, reducing AF load, and reducing the chance of AF occurrence or progression by targeting ion channels, inflammation, oxidative stress, and so on

The effects of excess accumulation has been reported and speculated to be negligible

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Ferroptosis was also implicated in the ALD development through the well-characterized crosstalk between the liver and other organs (e.g., gut, adipose, brain, and lung), among which the intestineliver and liveradipose interactions are most frequently reported [76]