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Oxidants/Antioxidants in the Liver and Lung of Individuals with Inherited Alpha-1 Antitrypsin Deficiency Severe inherited alpha-1 antitrypsin deficiency (AATD) is a monogenic, autosomal, co-dominant, inherited disease associated with a risk for developing liver disease at any age and/or early onset emphysema

For example, treatment with GS downregulated RANKL induced osteoclastogenesis and blocked IL-1beta mediated production of chemokines and epithelial neutrophil activating peptide-78 (ENA-78), MMP-1,-3 via suppression of NF-B, nuclear p50, and p65 subunit and IB degradation in rheumatoid arthritis (Ichikawa and Aggarwal, 2006

However, these side effects are typically mild and short-lived, and most patients do not experience any significant problems

These encouraging results warrant a larger clinical trial to confirm these findings

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