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doi: 10.1177/000348946207100313 63 SaundersG

They found that miR-200 targeted FOG2 which binds to p85 and negatively regulate PI3K activity suggesting that miR-200 regulates cell proliferation by regulating PI3K-AKT signaling activity

To further explore whether this dimerization process is a substrate-induced effect, we co-expressed another substrate ETS2, with the DDD-Ube2e2-COP1 complex and visualized the resulting assembly (Supplementary Fig