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The results illustrate the potential of a mechanism-guided therapeutic strategy by targeting a metabolic vulnerability in the ferroptosis of CSCs to improve the efficacy of lung cancer immunotherapy

Mitophagy deficiency increases NLRP3 to induce brown fat dysfunction in mice

Upon release into the circulation, these cytokines exert diverse effects on various tissues, such as the brain, muscle, and adipose tissues, leading to appetite suppression, suppression of muscular protein production, and promotion of lipolysis, which ultimately lead to the progression of cachexia

instead, the leading model holds that GHK-Cu delivers copper to intracellular pools and concurrently engages multiple signaling intermediates linked to fibroblast activation

Silencing p53 significantly alleviated PMB-induced iron accumulation and lipid peroxidation, increased the expression of TFR1 and ALOX12, and decreased the expression of GPX4 and SLC7A11 [178]