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Also, the co-administration of PA and APAP led to improvements in the pathological changes in the form of minimal congestion of the central vein

GCL activity and the expression levels of ferroptosis markers, GPX4 and acyl-CoA synthetase long-chain family member 4 (ACSL4), were remarkably improved in the REST -knockdown cells under the HR condition, rather than other ferroptosis-associated proteins (GCLC, dihydrofolate reductase [DHFR], and ferroptosis suppressor protein 1 [FSP1]), apoptosis-associated proteins (B cell leukemia/lymphoma 2 [Bcl-2] and Bcl-2associated X [Bax]), autophagy-associated proteins (microtubule-associated protein 1A/1B-light chain 3 [LC3] and sequestosome 1 [p62]), or pyroptosis-associated proteins (caspase-1 and GSDMD) (Figure 4D and Supplemental Figure 11, C and D)