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Properly controlled trials with structural and symptomatic outcome measures would establish whether BPC-157 provides disease-modifying effects or primarily symptomatic relief in joint disorders

GlyNAC supplementation corrected defective MFO (in mitochondrial fattyacid and glucose oxidation) but did not affect energy expenditure

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This hypothesis is further supported by clinical observations that more than 60% of accumulated fatty acids in the liver are derived from the circulation in NAFLD patients 7,8