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The present observations suggest that nano-chitosan priming may be associated with the modulation of antioxidant-related responses under salinity stress

Altogether, our work establishes SLC33A1-mediated GSSG export as a key mechanism for ER redox homeostasis and protein maturation

Transdifferentiation of EC via the process of endothelial-mesenchymal transition (EndoMT) and more likely of pericytes into profibrotic myofibroblasts may contribute further to vascular wall fibrosis [77]

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