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63 Prolonged skeletal muscle inactivity promotes mitochondrial dysfunction, resulting in increase in mitochondrial ROS production and leading to protracted oxidative stress and disturbed redox signaling, 64 which can exacerbate IR through multiple pathways such as JNK and NF-B pathways (Fig

10.1016/j.mcp.2023.101899 29 MiyamotoH

28 GSH carries a cysteine residue with an active thiol group and is responsible for maintaining thiol equilibrium