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doi: 10.4049/jimmunol.169.4.1837 108 ColeTJBlendyJAMonaghanAPKrieglsteinKSchmidWAguzziAet al

Radiation-induced oxidative stress has been demonstrated to induce endothelial dysfunction by reducing BH4 levels leading to the uncoupling of NOS in both the heart and lungs (162, 163, 182)

Although the suppressive function of ARG1 is well established, studies have shown heterogeneity in its expression and impact across tumor types and microenvironments

The PgAFP-F-S and AfAFP-F-S proteins exhibited a clean background on SDS-PAGE (Fig

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