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Marking experiments have shown that, in mutant strains with loss of adhesion auxiliary proteins, the P1 protein is chronically dispersed as a precursor in the cell membranes, however, it cannot aggregate to the apical organs or convert into mature P1 protein (9)

However, with the depletion of GSH in liver cells, reactive metabolites gradually accumulate, resulting in cytotoxicity, which eventually leads to liver cell necrosis and acute liver failure (Figure 3)

A reduced GSH/GSSG ratio was observed in the liver of hyperlipidemic rats (39% vs

All vitamin concentrations were statistically different between the control and HAD groups from the second (serum -tocopherol, plasma and leukocyte ascorbate