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Once the body is in oxidative stress, or if there are a large number of electrophiles or cytotoxic agents, Nrf2 is released from the Keap1 binding site and rapidly transferred to the nucleus, subsequently interacting with the antioxidant response element (ARE) in the promoter region of the target gene and then activates the transcriptional pathway to balance oxidative stress and maintain cellular redox homeostasis (Zhang, 2006)

Circulating levels of brain-derived neurotrophic factor: correlation with mood, cognition and motor function

Pituicyte cues regulate the development of permeable neuro-vascular interfaces

The mechanisms have potential for use in developing new pharmaceutical strategies to maintain HGF functions in many pathological conditions

It's largely anecdotal, based on the logical (but not necessarily scientifically validated) idea that putting the peptide closer to the site of injury will make it work better or faster