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JMF4073 eliminates WT-Bcr-Abl-transformed myeloid cells in vitro and in vivo Since the balanced supply of dNTPs is critical for overcoming DNA damage and replication stress for cell survival, we then tested whether JMF4073 treatment could selectively suppress the growth of Bcr-Abl-transformed but not untransformed myeloid cells

lower mitochondrial activity but increased glycolysis) that partially mirrors the metabolic properties of wild-type MZB

10.1161/CIRCRESAHA.116.309326 Circ Res

Notably, this metabolic transition is paralleled by nuclear remodeling, as indicated by the stronger hnRNP-E1 signal in iPSCs, which likely reflects their characteristically large nuclear volume, a hallmark of pluripotency

A modified SELEX approach to identify DNA aptamers with binding specificity to the major histocompatibility complex presenting ovalbumin model antigen