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Functionally, MAPKAPK2 overexpression mitigated oxidative damage, restored antioxidant capacity, and attenuated markers of DNA damage and cellular dysfunction, suggesting a cytoprotective role under oxidative conditions

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Future studies urgently need to address the following questions: What is the temporal sequence and causal relationship of various mitochondrial abnormalities (decreased biogenesis, dynamic imbalance, autophagic defects) during the long course of human PD