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I remembered there's a video someone posted a bit down in this thread link here:healthunlocked.com/pasoc/po

The clinically normal heterozygous carriers for this disease have about half of the normal glutathione synthetase activity, as predicted, but have normal clinical phenotypes, cellular glutathione levels, and 5-oxoproline excretion (53)

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P., Prasad, V

These effects are amplified when GHK-Cu is combined with procedures like microneedling or laser resurfacing that create controlled tissue injury and demand a repair response