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Chronic inflammation, seen in conditions like rheumatoid arthritis and inflammatory bowel disease, is both a cause and consequence of sustained oxidative stress [13]

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Overexpression of intestinal TXNIP abolishes the glucotonic effect of metformin TXNIP overexpression was induced to examine the reduction in metformin efficacy and to assess the replicability of TXNIP regulation in diabetes animal models

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The idea behind it is genuinely elegant: rather than tweaking brain chemicals the way caffeine or a stimulant does, dihexa was supposed to help neurons physically grow new synapses the connection points where brain cells talk to each other