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Performance enhancement at the cost of potential brain plasticity: neural ramifications of nootropic drugs in the healthy developing brain

Consequently, when considered collectively, these data suggest that hydrochlorothiazide could potentially induce glucose intolerance, particularly in mice models, through various mechanisms including those partially associated with -cell function and mass, those related to intermediary metabolism, alongside others yet to be uncovered

C., Makdani, D., Hegar, A., Rao, D., and Douglass, L

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In addition, it also downregulates Macrophage inflammatory protein-1 alpha (MIP-1) and Macrophage inflammatory protein-1 beta (MIP-1)