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Thus, further research into identifying reliable biomarkers is crucial for translating vascular normalization into widespread clinical use

Further simulations showed that over a wide range of doxorubicin (050 M), the model can be used to suggest the specific concentration and exposure duration required to deplete cytosolic GSH by 70%, the threshold by which antioxidant defence is impaired, protein binding occurs, and cell death is possible

BMC Genomics 25 (1), 32

We thank Donald Senger for help with Miles permeability assays, Myrtha Constant for technical assistance, and Howard Baden, Paolo Dotto, Paul Goetinck, and Ethan Lerner for helpful discussions and critical reading

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