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Exogenous H 2 S activated the nuclear factor erythroid 2-related factor 2 (Nrf2)/glutathione peroxidase 4 (GPx4)/glutathione (GSH) pathway, suppressed ferroptosis and mitigated mitochondrial apoptosis in db/db mice

All mice used in experiments were under a strict standard of care and experimental planning, covered by licensed approval from the Tel Aviv University Institutional Review Board and the Israeli Ministry of Health (License number 01-19-070)

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When glutathione production drops, you are more vulnerable to developing autoimmune disease, chronic pain, chemical sensitivities, leaky gut, and other immune-related disorders

Our study demonstrated that QCHS significantly alleviated DSS-induced ferroptosis in the colon of UC mice, and represents the first evidence linking L