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We bring forward the hypothesis that inflammation via prolonged activation of key kinases (p38 and GSK-3) and activation of histone deacetylases gives rise to dysregulation of the NRF2 system in the brain, which contributes to oxidative stress and injury

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Scand.J Clin Lab Invest 1985;45(2):169-178

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WangMLiuMWangCXiaoYAnTZouMet al

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