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It was found that a 23-fold -synuclein overexpression in hippocampal and ventral midbrain neurons impeded synaptic vesicle release [116] while -synuclein knock-out mice displayed stronger release of DA upon stimulus [117]

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Likewise abnormalities in potassium channel have been reported in neurodegenerative diseases including amyotrophic lateral sclerosis (ALS), a lethal neurodegenerative disease commonly called Lou Gehrigs disease 30 , and Parkinsons disease such that potassium channel blockers are part of treatment regimen in Parkinsons disease 31

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Complement complexes can be discovered in atherosclerotic lesions, and ECs exposed to complement are activated and improves the production of IL-6, IL-8 and MCP-1, thereby triggering the adhesion of monocytes to ECs 89