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68 Type I IFNs have also been demonstrated to suppress tumor angiogenesis by regulating the expression of various pro-angiogenic cytokines, including MMP9, VEGF, and a range of CXCL chemokines (CXCL1, CXCL2, CXCL5, and CXCL8)

While acetylation of this residue is usually found in active chromatin (Peters et al., 2001

Conversely, ADAR1 overexpression promotes apoptosis in U87 and HepG2 cells, highlighting its role in modulating cell death through post-transcriptional regulation of antiapoptotic genes [175]