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In vitro experiments demonstrated that agrin treatment significantly downregulated PDGFR and claudin-5 expression in a blood-brain barrier co-culture model comprising human brain microvascular endothelial cells and pericytes, suggesting that agrin may induce pericyte death and impair the integrity of the blood-brain barrier

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The increased oxidative environment resulting from NOX activity and mtROS production leads to GSH depletion

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Dyslipidemia is very common in patients with diabetes [20]