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doi:10.1007/s00441-022-03579-z 1098. Qiu CP, Lv QT, Dongol S, Wang C, Jiang J

proposed a hypothesis in a mouse model, suggesting oxidative stress in livers with preexisting fatty liver disease might play a role

Moving from proof-of-concept preclinical research to rigorous translational pipelines that incorporate standardized formulations validated biomarkers phenotype-specific patient selection and clinically significant reproductive and metabolic outcomes is the most urgent need

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It was found that the expression of GCH1 triggers the production of potent antioxidants BH4/BH2, which can block lipid peroxidation and ferroptosis, whether supplemented exogenously or produced endogenously