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Bryostatin-1 attenuates intestinal ischemia/reperfusion-induced intestinal barrier dysfunction, inflammation, and oxidative stress via activation of Nrf2/HO-1 signaling

The main difference between these two mechanisms is that active targeting utilizes interactions between nanoparticles and over-expressed receptors on cancer cells and vascular endothelial cells, thereby enhancing nanoparticle accumulation in the tumour