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When it binds there, it reduces neuroinflammation, supports nerve fiber regrowth, and turns down the inflammatory signals that keep nerve pain going long after the original cause has resolved

Kara Fitzgerald: Yep, absolutely

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Relationship between the clinical state and intrathyroidal iodine as measured in vivo in man

PPAR- is a nuclear receptor that regulates different metabolic pathways and inflammatory responses, where activation of this receptor can interfere with TGF-1 signaling via the Smad pathway and suppress the production of inflammatory mediators by downregulating NF-B