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For biotechnological applications, high cell densities are desirable, but metabolic limitations are more likely to arise under these conditions, even in continuously fed systems (fed-batch, semi-continuous and continuous reactors), putting cell viability at risk

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Some studies have demonstrated that -2 adrenergic agonists can effectively counteract the accelerated muscle protein catabolism observed in cancer cachexia models, including myocardium and skeletal muscles, 153,154 suggesting the potential role of SNS activity in modulating skeletal muscle wasting during cancer cachexia

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