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Collectively, ferroptosis plays a significant role in the pathogenesis of bacterial infections by contributing to host tissue damage and influencing immune responses

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To achieve this, understanding the formation mechanism of DTP cells and their resistance-related properties provides a basis for subsequent therapeutic management

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As MoAMs are the culprit cells responsible for promoting lung fibrosis, genetic/epigenetic modulation holds promise for precise targeting while sparing the homeostatic function of the resident macrophages