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aeruginosa biofilms inhibiting aminoglycoside-induced adaptive resistance (Maiden and Waters, Conclusion This study adopts GC-MS-based metabolomics to explore CIP metabolic resistance mechanisms

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Despite distinct primary targets, both converge on inhibiting acetyl-CoA carboxylase (ACC1), elevating acetyl-CoA levels, preserving mitochondrial homeostasis, and ultimately blocking ferroptotic cell death

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Notably, the differences in DPPH inhibition among EtOAc, MeOH, H 2 O, and 70% EtOH mycelial extracts were minimal for both macrofungi cultivated in the GPYB medium but more pronounced in those grown in the SDB medium