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oral GSH administration sensitized healthy and diabetic individuals to insulin [6]

Lack of glutathione peroxidase-1 exacerbates a beta-mediated neurotoxicity in cortical neurons

Chronic aspartame intake causes changes in the trans-sulphuration pathway, glutathione depletion and liver damage in mice

Most commonly, oligodendrocyte differentiation abnormalities are associated with disruption of oxidative stress phenomena associated with excitotoxicity, mediated by glutamatergic metabotropic receptors in large amounts in oligodendrocyte precursors [22, 137]

However, relatively high glutathione levels in the periplasm of E