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Te veel reactieve zuurstofsoorten kunnen de werking en de structuur van cellen verstoren, maar levende organismen kunnen niet zonder

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The hepatocyte-specific deletion of the endogenous inhibitor of RIPK1 kinase, such as TAK1- which performs inhibitory phosphorylation on RIPK1- results in spontaneous hepatocyte apoptosis, steatohepatitis, fibrosis, and HCC 67,68