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However, an emerging conceptual shift suggests that lipid peroxidation and ferroptosis significantly contribute to FRDA pathogenesis, acting in concert with other well-established disease mechanisms such as mitochondrial bioenergetic failure, apoptosis, and inflammatory signaling [29,30]

In TM cells, after exogenous oxidative treatment, the following damage has been observed: a decrease in cellular activity (102), a change in cell cycle progression, the inhibition of cell proliferation (103), and the promotion of cellular senescence (100)

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Additionally, H19, a significant lncRNA, regulates multiple signaling pathways in liver and colorectal cancer, affecting tumor cell proliferation and survival (11)

Improved liver glutathione status means more efficient toxin processing, supporting your body's natural ability to eliminate environmental pollutants, pharmaceutical metabolites, and cellular waste products that would otherwise accumulate and contribute to oxidative stress