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The convergence of synaptic dysfunction, mitochondrial failure, neuroinflammation, and impaired protein clearance underscores the complexity of the disease and the need for early, multi-mechanistic neuroprotective interventions

A dual-targeting strategy focused on lactate metabolism and lactylation regulatory enzymes offers the potential to simultaneously address immunosuppression and drug resistance, establishing a novel method for precision lactylation intervention

Furthermore, they can also be more easily operated than MS in regions with unreliable power supply