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Synthesis of ketone bodies may be initially increased in MASLD as a mechanism to safely manage excess triglyceridederived acetylCoA via nonoxidative disposal to limit oxidative stress, a mechanism which becomes disturbed as MASLD progresses (Post et al

Inhibitors of these adaptors can be considered as potential therapeutic agents in asthma

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Damaged hepatocytes release a variety of chemokines [163, 164] and DAMPs [165, 166], which promote immune cell infiltration in ALD

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