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Description

In contrast, IL-4-stimulated macrophages exhibit increased OxPhos, a process dependent on PPAR-coactivator-1 (PGC1)-mediated mitochondrial biogenesis, CD36-dependent lysosomal lipolysis, and fatty acid oxidation (FAO) (Fig

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The NFE2L2 (Nrf2) pathway constitutes a key cellular defense against ferroptosis

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