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7,8 Although cellular senescence plays a physiological role in development, its associated pathological changes, such as chronic low-grade systemic inflammation, cartilage thinning, reduced glycosaminoglycan content, altered proteoglycan composition, accumulation of advanced glycation end-products, impaired autophagy, and apoptosis, are key drivers of OA onset and progression

Similarly, doped and polymer-based CQDs, including nitrogen-, copper-, and poly (lactic-co-glycolic acid)-doped systems, have shown improved stability, synergistic fluorescence, theranostic capabilities [91, 182]