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[DOI] [PMC free article] [PubMed] [Google Scholar] 36.Camponeschi A, Klasener K, Sundell T, Lundqvist C, Manna PT, Ayoubzadeh N, Sundqvist M, Thorarinsdottir K, Gatto M, Visentini M, Onnheim K, Aranburu A, Forsman H, Ekwall O, Fogelstrand L, Gjertsson I, Reth M, and Martensson IL

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In agreement with earlier research, our findings demonstrated that abemaciclib triggers the endoplasmic reticulum (ER) stress response, modifies the mitochondrial membrane potential, and consequently causes loss of membrane integrity, cytochrome c release, ROS generation, cellular endogenous antioxidant depletion, caspase activation, pro-apoptogenic proteins BAX and P53 upregulation, and anti-apoptogenic protein Bcl-2 downregulation