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Furthermore, wound-healing studies published in Life Sciences [4] indicate that copper-peptide complexes accelerate repair while reducing oxidative markers of inflammation

Unlike classical inflammation (characterized by heat, redness, and pain), neuroinflammation primarily involves the release of pro-inflammatory mediators (e.g., cytokines, chemokines, and ROS) by resident immune cells, particularly microglia, the CNSs innate immune effectors [110, 111]

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