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Necroinflammation and fibrosis are important intermediary steps prior to developing cirrhosis and they involve alterations in hepatic parenchymal and non-parenchymal cell (NPC) populations

The relative abundance of arsM in prophages declined from (3.3 0.2)E-2 copies/VLP on day 0 to (2.0 0.1)E-3 copies/VLP on day 1 (Fig

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GPX4-IN-3 (26a) is more likely to induce ferroptosis through the accumulation of intracellular peroxides via inhibiting GPX4 activity [1]