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After TBI, there is an activation of neuroinflammation response pathway mediated by microglia cells that were not initially damaged, and the activated microglial cells may promote the release of proinflammatory factors such as interleukin 1, tumor necrosis factor, nitric oxide, etc., (Kumar and Loane, 2012), inducing a progressive cycle of chronic inflammation (Sulhan et al., 2020)

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Key limitations: Some compounds have direct human pharmacodynamic data

The study determined that from Enterococcus faecalis tyrosine decarboxylase can decarboxylate L-dopa to dopamine, and that Eggerthella lenta dopamine dehydroxylase can further convert dopamine to m-tyramine

Thus, we aim to evaluate the effectiveness of psychosocial therapy in patients with chronic liver disease (CLD) with AUD