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Circulating IGFBPs, especially IGFBP-3 and IGFBP-5, can combine with circulating IGFs and acid-unstable subunits produced by liver cells to create terpolymer complexes around 140 kDa, resulting in a prolonged half-life of IGFs in circulation that can slow down the movement of IGFs to tissues outside of blood vessels [89, 90]

Secreted CLIC3 drives cancer progression through its glutathione-dependent oxidoreductase activity

The first clinical trial of ambroxol in PD demonstrated its ability to cross into the cerebrospinal fluid and increase glucocerebrosidase protein levels regardless of the presence of GBA1 mutations [180]

In ALD, this bidirectional crosstalk is further amplified, wherein iron overload contributes to microbial dysbiosis and intestinal barrier dysfunction, while microbiota-mediated alterations in iron absorption through HIF-2 pathway aggravate hepatic injury

Y., Yun, J., Wagner, T