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6F and G, treatment with blank NPs, ATRA, or ATO did not result in significant downregulation of PIN1 and its substrate oncogenes including -catenin, NF-B and Cyclin D1 in tumors compared to the saline control

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In this model, reduced phosphorylation of Drp1 and altered proteolytic processing of Opa1 led to increased mitochondrial fission, suggesting that increased lipid uptake, as typically observed in diabetic hearts, may trigger mitochondrial fragmentation and thus mitochondrial dysfunction and ROS [45]