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This is evident from the reduction in electrographic seizure-like activity in transgenic mice overexpressing neuronal adenosine kinase or lacking the adenosine A1 receptor [112]

These observations were also made with mechanistically different secondary stressors such as the mitochondrial complex I inhibitor, rotenone, or proteasomal inhibitors such as MG-132 or bortezomib (Suppl

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NLCs were designed specifically to address some limitations of the first-generation SLNs- such as limited loading capacity and the tendency of some active compounds to be expelled from the solid lipid matrix during storage

However, it is crucial to consult with healthcare professionals to develop a personalised management plan based on individual needs.