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They found that DDD therapeutically targets AS by enhancing mitochondrial function through the HIF-1 signaling pathway, reducing ROS levels, and modulating GPX4, Bcl2, and Bax protein levels, thereby benefiting AS and hyperlipidemia (Wu et al., 2023a)

Researchers often examine it in models involving neuronal signaling, synapse formation, and learning-related pathway behavior

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Researchers study GHK-Cu because it is relevant to extracellular-matrix, signaling, and cellular-response models in controlled experimental systems