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These research findings prompt us to propose the hypothesis that both ferroptosis and disturbances in cysteine metabolism might constitute significant mechanisms in the pathogenesis of AA

The mechanism by which Zn inhibits iNOS is by limiting the nuclear factor (NF)-B transactivation activity

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CMT4B3 fibroblasts showed a comparable pattern, with no significant effect at 0.1 mM followed by inhibition of approximately 73% at 1 mM and 82% at 5 mM (Fig