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This review summarizes the role of these two signaling pathways in intestinal diseases and explores the interconnections between them (see Figure 1)

This approach aims to overcome the bioavailability challenges associated with TQ and unlock its full therapeutic potential in managing psoriasis (4, 7)

observed abundant autophagic vesicles and enhanced autophagic flux in SCs from the HG group (90), autophagy hyperactivity was also observed in the streptozotocin (STZ)-induced rat model of DPN in Wang et al., these effects were also found to potentially increase apoptosis in SCs and demyelination of sciatic nerves in rats with DPN (91)

Medication may reduce nerve excitability, but pain can continue if there is ongoing vascular compression, peripheral branch irritation, dental-related nerve trauma, postherpetic nerve injury, muscle spasm, scar tissue, local inflammation, or central sensitization

Cancer-associated fibroblasts build and secure the tumor microenvironment