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Therefore, in addition to L-carnitine for mitochondrial fatty acid transport, ALCAR provides acetyl-CoA that can be used for brain lipid synthesis, be oxidized in the citric acid cycle for energy production, or be incorporated into neurotransmitters such as acetylcholine [3639].* ALCAR has shown several benefits for brain health: it supported brain energy metabolism, brain mitochondrial function, neuronal membrane lipid/phospholipid metabolism, synaptic plasticity, and neuroprotective functions [4056]

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ERK1/2 activation has been reported to regulate cytokine production, epithelial cell responses, airway smooth muscle proliferation, and immune cell activation in the asthmatic airway microenvironment (Makled and El-Sheakh 2023)

As we get older, we make less catalase

In contrast, the detrimental effect of mitophagy mostly occurs in scenarios of excessive activation or abnormal regulation: when cells encounter sustained and severe pathological stress (such as severe ischemia and severe inflammation), mitophagy is abnormally activated, which excessively degrades functionally normal mitochondria, leading to disorders of cellular energy metabolism, insufficient ATP synthesis, and simultaneous disruption of the normal proliferation and renewal balance of mitochondria in cells