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Adipose triglyceride lipase contributes to cancer-associated cachexia

Extrapolating from cancer cells performing -oxidation under hyperoxic conditions in vitro to assuming they do so in vivo is akin to judging how fast a fish can run on land by measuring how swiftly it swims in water: the environment fundamentally alters the outcome, misrepresenting the true metabolic behavior of cancer cells within a living organism

doi: 10.3390/antiox13080985 37

213,214 Specifically, disturbances in the circadian rhythm not only enhance the malignant potential of breast cancer cells (including their ability for self-renewal, replication, metastasis, and invasion) but also impact chemokine/chemokine receptor signaling (the CXCL12-CXCR4 axis may be the primary signaling pathway) which contributes to the formation of an immunosuppressive microenvironment ultimately leading to tumor progression