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Thus, heterosynaptic changes in inhibitory neurons were weight-dependent: Inputs with initially high paired-pulse ratio (low release probability, weak inputs) tended to be potentiated while inputs with initially low paired-pulse ratio (high release probability, strong synapses) tended to depress or did not change after intracellular tetanization

For example, changes in both the presynaptic adhesion molecule neurexins and the postsynaptic adhesion molecule neuroligins have been observed to be associated with changes in synaptic plasticity in early life stress, stressed-sedentary, Parkinsons disease, and Alzheimers disease [54,55,56,57,58]

S., & Ponesakki, G